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Sentences for "antagonism"
Tannen's antagonism toward antagonism makes sense in the former case but not in the latter.
So let us stop this campaign of antagonism towards Italy and start using our resources and friends in Italy to convey our clear messages to the masses.
In his response to Bush's complaint Exley rehearses the history of Bush's antagonism toward his site, citing W.
The mechanism of RGS action is antagonism of the G?
protein either by accelerating its hydrolysis of GTP, thereby prematurely terminating signaling due to reassociation of the G?
and Gß?
complexes [ 10 ] , or by antagonizing interaction of the G?
protein with its effector such as phospholipase Cß [ 11 12 ] .
Finally, the functional antagonism between TGF-ß1 and Hyal-1 or Hyal-2 in regulating TNF susceptibility in L929 cells was examined.
These data indicate that there is a functional antagonism between TGF-ß1 and Hyal-1 or Hyal-2.
Interestingly, Wnt-5a down-regulated mesothelin expression, perhaps through antagonism of endogenous Wnt/beta-catenin signaling.
The extent to which these factors may regulate BMP-5 signaling is difficult to determine because of the paucity of data regarding either antagonism of BMP-5 signaling by follistatin and noggin or the interaction between known BMP receptors and BMP-5.
(3) These in vitro data can be utilized to define the physiochemical features of the ligands defining agonism and antagonism at ?
opioid receptors, leading to design and discovery of novel opioid analgesics.
Selective antagonism of D2 receptors only modestly reduced the amplitude of HFO without affecting any other urodynamic parameters [ 6 35 ] .
Therefore, it is possible that olanzapine and risperidone decrease the amplitude of the HFO, at least partly, through antagonism of D2 receptors.
Both olanzapine and risperidone have moderate affinity for the alpha 1 adrenergic receptor (Table 1), therefore it is possible that some of the effects observed in the present study were due to central and/or peripheral antagonism of alpha 1 adrenergic receptors by these drugs.
In cats, serotonin antagonists caused a decrease in bladder capacity [ 43 ] however in anesthetized rats, serotonin antagonism did not have an effect on micturition [ 44 ] .
Serotonin 5-HT2 and 5-HT3 receptors facilitate pudendal reflexes in the cat [ 47 48 ] , therefore it is possible that antagonism of these receptors may block the EUS in the anesthetized rat.
Since clozapine crosses the blood-brain barrier readily [ 14 ] , the maximum dose selected in the present experiment was restricted to the first appearance of a decrease in blood pressure as an indication of possible peripheral alpha 1 antagonism due to peripheral leakage.
Spinal antagonism of either alpha1 or alpha2 adrenergic receptors was reported to increase micturition volume [ 17 ] in anesthetized rats.
Therefore, it is difficult to interpret clozapine's effects on micturition volume in terms of spinal alpha adrenoceptor antagonism and possibly the supraspinal effects predominate.
A decrease in residual volume has been reported following spinal antagonism of alpha2 receptors with yohimbine [ 17 ] but atipamezole produced an increase in the residual volume [ 18 ] , similar to our findings with clozapine i.
Thus, although alpha1 antagonism has been shown to modulate pudendal reflexes in the cat, their role in modulating the activity of the EUS during micturition in the rat appears unclear.
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