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Sentences for "atherosclerosis "
txt Background Atherosclerosis and coronary heart disease (CHD) have long been considered major contributors to morbidity and mortality in developed countries [ 1, 2, 3].
PPARs are important in atherosclerosis but the effect of PPAR activation on cholesterol oxide induced PCD has not been determined, this study was designed to test if a dysregulation of MAP kinases was associated with the cholesterol oxide induced PCD, and to determine if PPAR activation could modulate the cholesterol oxide induced PCD.
Hypercholesterolemia primarily during childhood, accelerated atherosclerosis in some adult patients leading to premature death, as well a hemolytic anaemia, arthralgias and tendon and tuberous xanthoma formations are other clinical features.
However, it is not known whether the atherosclerosis in sitosterolemic patients with hypertension is accelerated, relative to those affected individuals with lower blood pressures.
They completed a questionnaire on risk factors for atherosclerosis including family history of coronopathy, smoking, high blood pressure, personal history of coronopathy or myocardial infarct, history of cerebrovascular disease or occlusive peripheral disease.
Shifting the object of investigation from ischemic stroke to carotid atherosclerosis would decrease the numbers of subjects needed.
Another advantage is that carotid atherosclerosis can be treated as a quantitative trait.
However, using carotid atherosclerosis as an intermediate phenotype for ischemic stroke also has limitations.
Perhaps the most important difficulty is that high-grade carotid atherosclerosis is not a prerequisite intermediate phenotype in most patients with ischemic stroke.
In addition to not being a mandatory intermediate phenotype in the biological process leading to ischemic stroke, carotid atherosclerosis is itself a complex phenotype.
Degree of stenosis is not the only factor affecting symptomatology of an atherosclerotic carotid artery [ 77 ] , and important differences between intracranial and cervical arteries in the genetics of atherosclerosis may go unrecognized if the object of study is cervical carotid atherosclerosis [ 78 ] .
[ 11 ] observed an association between carotid atherosclerosis and the C148T polymorphism in a population-based cross-sectional study of persons with normal neurologic status.
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