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Sentences for "autoantibody "
We used one-tailed tests to look for an increase in autoantibody as compared to controls and we set P < 0.
We therefore were interested in finding out how IFN-?
and IL-4 regulate isotype expression of the PG-specific antibodies and if an autoantibody isotype contributes to the severity of disease.
Conclusion Th1 and Th2 cytokines regulate PG-specific autoantibody IgG isotypes and susceptibility to arthritis.
The correlation between PG-specific autoantibody isotype and development of arthritis suggests a pathological role for IgG2a isotype in disease.
This has been achieved primarily by expression cloning using human autoantibody probes.
The emerging view is that modified forms of autoantigens generated during cell death might stimulate autoantibody responses if presented to the immune system in a proinflammatory context [ 23 ] .
Since the cleaved Golgi autoantigen fragments are antigenic, they may play a role in sustaining autoantibody production in autoimmune disease states.
Role of apoptotic or necrotic fragments in autoantibody production Analysis of post-translational modifications in intracellular autoantigens associated with apoptosis and necrosis should provide important insights into the possible role of cell death in the generation of potentially autoantigenic forms of cellular antigens.
Unique proteolytic fragments of ?
-fodrin and type 3 muscarinic acetylcholine receptor have been implicated in the generation of autoantibody responses in primary Sjögren's syndrome [ 38 39 ] .
Although direct evidence that autoantigen cleavage fragments produced during cell death elicit autoantibody responses is still scarce, Pollard et al .
These data point to a crucial role for fragments of autoantigens in the generation of autoantibody responses.
We also speculate that Golgi fragmentation in cell death might participate in sustaining autoantibody response to the Golgi complex in aberrant disease states characterized by unregulated cell death resulting from injury or infection or defective clearance of dying cells.
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