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Sentences for "autoimmune "
[ 4 ] For many patients with autoimmune disease the target antigen(s) is unknown.
A human autoimmune serum against a nucleolar antigen ANA-N was used for nucleolar co-localization (data not shown).
The phenotype of the Blau syndrome resembles another multisystem granulomatous disorder, Crohn's disease (MIM 266600), and another autoimmune disease, psoriasis (MIM 177900).
Interestingly, another autoimmune disease ankylosing spondylitis (MIM 106300), also maps to chromosome 16q, however, it appears to map telomeric to the Blau syndrome interval [ 9 10 ] .
Behçet's disease is an autoimmune disorder characterized by a systemic vasculitis that affects the joints, all sizes and types of blood vessels, the lungs, the central nervous system, and the gastrointestinal tract [ 38 ] .
These genes may also serve as candidates for other autoimmune disorders that map to this region such as IBD3.
These findings assume clinical importance since they may explain in part the chronic and autoimmune pathologies in the elderly, particularly rheumatoid arthritis, Crohn's disease, sepsis, and myelodysplastic syndromes, which are exacerbated by the inflammatory cytokines IFN-?
and TNF-?
[ 41 ] .
Interestingly, both IFN-?
and TNF-?
are prominent in the pathogenesis of autoimmune diseases in general, the former by activation of local auto antigen presentation to appropriate T cells, and the latter by mediation of inflammatory tissue damage [ 68 ] .
It is logical to infer that the increased susceptibility of aged individuals to rheumatoid arthritis and other autoimmune pathologies, may be due to increased TNF-?
(and IFN-?
) production by the CD8 +CD28 -T cell.
Thus the advent of the CD8 +CD28 -cell in aging, with its potential for proinflammatory cytokines, could have profound effects not only on autoimmune diseases, but on Alzheimer's disease as well since cytokines may contribute to the neuropathology [ 72 ] .
txt Background Type 1 diabetes mellitus (T1DM) is a T cell-mediated autoimmune disease characterized by lymphocytic infiltration of the pancreatic islets of Langerhans with subsequent destruction of the insulin-producing beta cells [ 1 ] .
Accordingly, a shift of the autoimmune response from T H 1 to T H 2 predominance has represented a promising strategy for prevention of diabetes and other T H 1-mediated autoimmune diseases.
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