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Sentences for "carcinomas "
Mis-regulation of Wnt signaling leads to the development of several human cancers, including colon carcinomas and melanoma [ 1 ] .
The soluble portion of mesothelin has drawn attention as a potential serum tumor marker since it is highly over-expressed in patients with ovarian carcinomas and pancreatic adenocarcinomas [ 27 28 ] .
Interestingly, mesothelin expression is induced in lung, ovarian, and pancreatic carcinomas where constitutive activation of Wnt signaling is caused by stabilizing mutations in beta-catenin [ 27 28 41 42 43 44 ] .
The induction of mesothelin in these carcinomas may be due to misregulation of Wnt signaling as are other targets of the Wnt pathway, c-myc and cyclin D1.
The PTEN gene is also frequently deleted or mutated in a variety of sporadic human malignancies, such as glioblastoma and carcinomas of the prostate, endometrium and breast.
Female mice harboring the Wnt-1 transgene develop extensive mammary hyperplasia early in life, and 50% of them develop mammary ductal carcinomas by 6 months of age [ 27].
After a mutant allele of Pten was crossed into Wnt-1 TG mice, we found that ductal carcinomas appeared earlier than either alteration alone and that LOH at the Pten locus occurred in the majority of these tumors, suggesting a selective growth advantage in cells that lack Pten functions.
These two carcinomas were serially passaged in syngeneic rats for more than 50 passages.
Results Tumour characteristics Carcinomas of the lung were chosen, as it is the most frequent tumour in the cancer population.
The angiogenesis in non-small-cell lung carcinomas in patients shows four different patterns of vascularisation: basal, diffuse, papillary and alveolar [ 29 ] .
The basal and the diffuse are the most frequent patterns found in squamous carcinomas and adenocarcinomas respectively.
We tested in this investigation the hypothesis that M6P/IGF2R loss of heterozygosity in locally advanced, non-metastatic squamous cell carcinomas of the head and neck is associated with poorer patient prognosis.
However, our previous finding, that both alleles of the M6P/IGF2R are mutated in greater than 50% of squamous cell carcinomas of the lung [ 22 ] , supports our postulate that the M6P/IGF2R is a key head and neck cancer tumor suppressor gene at chromosome location 6q.
Furthermore, since M6P/IGF2R loss of heterozygosity in squamous cell carcinomas is highly correlated with an elevated plasma TGFß level [ 39 ] , patient prognosis may potentially be assessed by a simple blood test.
The cyclooxygenase pathway and NSAIDs have been implicated in pancreatic carcinogenesis, as elevated levels of COX-2 mRNA and protein have been detected in pancreatic carcinomas relative to histologically normal cells [ 14 15 16 17 18 19 ] .
29 more results not shown.
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