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Sentences for "cochlear "
The first set of genes, transmembrane cochlear expressed genes 1 and 2 ( TMC1 ) and TMC2 [ 5 ] were found in a search for mutations causing dominant and recessive deafness in human and mouse [ 5 6 ] .
Bth / Bth mice display altered potassium currents in early postnatal cochlear hair cells, in particular Ik,n and Ik,f currents appear to be depressed [ 10 ] .
Conclusions The recently identified genes encoding the cochlear transcripts for TMC1 and TMC2, and encoding the proteins EVER1 and EVER2, belong to a novel gene family that is conserved in animals.
In fact, the selective loss of vestibular as compared to cochlear sensory neurons (77% versus 29%, [ 3 ] ) suggests that NT-3 expression may be less downregulated in Brn3c null mutants than BDNF [ 6 8 9 ] , provided that at least some differentiation of supporting cells takes place.
We report here long term retention of cochlear sensory neurons for at least 6 months, in particular in the cochlear apex, in Brn3c null mutant mice.
Comparison of numbers of immature hair cells in the vestibular and cochlear areas (Fig.
4) with the numbers of surviving vestibular and cochlear sensory neurons does not show any correlation (Table 1, Fig.
And yet only about 50% of cochlear sensory neurons are lost overall with a less severe loss in the apex in which we could not detect any immature hair cells with MyoVIIa immunocytochemistry.
The central projection of DiI and DiA traces fibers to the cochlear nuclei (Fig.
6, red) in both the entering cochlear nerve (Fig.
6a) and the ventral cochlear nucleus (VCN) (Fig.
Qualitative comparable central projections are found in wildtype control littermates (data not shown) and suggest that the central projection to cochlear nuclei is at least until P8 in mice not critically dependent on auditory information or other hair cell-mediated influences, in agreement with recent suggestions [ 5 19 ] .
The comparatively high level of NT-3 expression in Brn3c null mutant cochlea, which has been shown to be the neurotrophin most prominently supporting spiral sensory neurons [ 6 9 14 ] , is in agreement with the normal development of cochlear innervation in newborn Brn3c null mutants.
Our data show that at least a cochleotopic projection in the cochlear nerve and the cochlear nuclei develops even in Brn 3c null mutants (Fig.
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