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Sentences for "conduction "
The natural processes result, on the one hand, in the preparation of fit and proper nutriment out of the materials of the digested food, for the growth and support of the several tissues and functions; and, on the other hand, in the gradual change and conduction out of the body of the products of the waste of those tissues.
The nerves are not naturally constituted for the conduction of medicinal impressions, but only for the passage of impulses of volition, sensation, or emotion.
txt Background Atrial fibrillation is a chaotic atrial rhythm resulting from abnormal signal generation and conduction in the atria.
gov/omim/) [ 44 ] , apolipoproteins E (MIM 107741), CII (MIM 207750) and CI (MIM 107710), human brain-specific Na(+)-dependent inorganic phosphate cotransporter (hBNPI, 19q13) [ 45 ] , cardiac troponin I (TnIc, 19q, MIM 191044) [ 46 47 ] , cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL, caused by mutations in the Notch3 gene, 19q12, MIM 602275) [ 48 ] , isolated cardiac conduction disease (19q13.
One of the striking characteristics from this family was the degree of cardiac conduction system disease that primarily targeted the atrioventricular node [ 1 10 ] .
In addition to DCM with conduction system disease and variable skeletal muscle involvement, mutations in the Lamin A/C gene [OMIM 150330] have been shown to cause autosomal dominant [ 11 ] and autosomal recessive [ 12 ] Emery-Dreifuss Muscular Dystrophy (EDMD), autosomal dominant limb-girdle muscular dystrophy type 1B (LGMD) [ 13 ] , autosomal recessive type 2 Charcot-Marie Tooth type 2 [ 14 ] , familial partial lipodystrophy [ 15 16 ] , Mandibuloacral dysplasia [ 17 ] and Hutchinson-Gilford progeria [ 18 ] .
Discussion Our findings demonstrate a novel mutation in lamin A/C in a proband whose family was previously linked to DCM with conduction disease and chromosome 1q21.
A recent report describing another family with a LMNA mutation causing DCM and conduction system disease documents the need for permanent pacemaker implantation [ 21 ] .
Interestingly, the preferential involvement of the specialized cells of the cardiac conduction system may support less the stress hypothesis as the cells of the atrioventricular node are less expected to undergo considerable deformation.
This indicates the necessity for careful documentation of a three-generation pedigree in the evaluation of patients with conduction system disease paying particular attention to sudden death, arrhythmia and bradycardia.
Genotyping of patients with conduction disease and DCM may provide important information regarding device implantation.
Conclusions Individuals with DCM and conduction system disease should be screened by family history and genetic testing.
Cardiac involvement in mitochondrial diseases Inborn errors of mitochondrial fatty acid oxidation should be considered in infants with conduction defects or ventricular tachycardia.
Cardiac conduction disturbance has also been recognized in the mitochondrial cytopathy, Kearns-Sayre syndrome (Mendelian inheritance in Man database *530000), characterized as progressive external ophthalmoplegia and retinopathy [ 16 ] .
The 1° afferents possessed higher conduction velocities (CV = 68.
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